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Keywords = HTN

  • Open Access Case Report
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    Trends Journal of Sciences Research 2018, 3(4), 147-150. http://doi.org/10.31586/Surgery.0304.01
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    Abstract
    Central venous devices are routinely used in delivering chemotherapy and total parenteral nutrition. Spontaneous migration of central venous catheters is a very rare complication, but the etiology of this problem is not clear. We report here a case of migration of a port catheter to the anterior mediastinum in
    [...] Read more.
    Central venous devices are routinely used in delivering chemotherapy and total parenteral nutrition. Spontaneous migration of central venous catheters is a very rare complication, but the etiology of this problem is not clear. We report here a case of migration of a port catheter to the anterior mediastinum in a patient with stage IVC nasopharyngeal cancer during chemotherapy. The patient presented with pulmonary manifestations in form of shortness of breath and chest tightness caused by left massive pleural effusion. The pleural effusion was resolved by thoracocentesis and the migrated catheter was retrieved surgically.  Full article
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    References
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    MC Lin, TK Chang, YC Fu, SL Jan. A magic port-A-cath. JACC Cardiovasc Interv 2013;6:e17-e18
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    WC Fan, CH Wu, MJ Tsai, YM Tsai, HL Chang, JY Hung, PH Chen, CJ Yang. Risk factors for venous port migration in a single institute in Taiwan. World J Surg Oncol 2014;12:15.
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    M Shah, S Patni, R Bagarahatta. Spontaneous chemoport fracture and cardiac migration. Indian J Surg Oncol 2014;5:325-326. doi:10.1007/s13193-014-0353-0.
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    BL Houston, M Yan. Spontaneous migration of an implanted central venous access device into the ipsilateral jugular vein. CMAJ2016;188:752.
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    KS Ahn, K Yoo, IH Cha, TS Seo. Spontaneously migrated tip of animplantable port catheter into theaxillary vein in a patient with severecough and the subsequent intervention to reposition It. Korean J Radiol 2008;9(Suppl):81-84. doi:10.3348/kjr.2008.9.s.s81
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    SN Nagel, UK Teichgraber, S Kausche, A Lehmann. Satisfaction and qualityof life: a survey-based assessment in patients with a totally implantablevenous port system. Eur J Cancer Care (Engl) 2012;21:197-204.
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    CY Wu, JY Fu, PH Feng, YH Liu, CF Wu, TC Kao, SY Yu, PJ Ko, HC Hsieh. Risk factors andpossible mechanisms of intravenous port catheter migration. Eur J VascEndovasc Surg 2012;44:82-87.
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    SL Yeste, JM Galbis Caravajal, CA Fuster Diana, EE Moledo. Protocol for theimplantation of a venous access device (Port-A-Cath system): thecomplications and solutions found in 560 cases. Clin Transl Oncol 2006;8:735-741.
  • Open Access Research Article
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    Trends Journal of Sciences Research 2019, 4(1), 21-28. http://doi.org/10.31586/Cardiology.0401.04
    74 Views 95 Downloads PDF Full-text (793.318 KB) PDF Full-text (794.026 KB) PDF Full-text (793.965 KB)  HTML Full-text
    Abstract
    At present, cardiovascular diseases are global health problems responsible for 17.3 million deaths per year and adding extra burden in developing countries like Nepal. Studies show that serum uric acid (SUA) can result in endothelial dysfunction which can lead to vascular disease like stroke. In this study, we determined serum
    [...] Read more.
    At present, cardiovascular diseases are global health problems responsible for 17.3 million deaths per year and adding extra burden in developing countries like Nepal. Studies show that serum uric acid (SUA) can result in endothelial dysfunction which can lead to vascular disease like stroke. In this study, we determined serum uric acid levels in patients with acute coronary syndrome (ACS) and assess its risk factors. A cross sectional study was conducted in 82 patients with ACS who fulfilled the inclusion criteria included in the study and their serum uric acid level were investigated. It was found that 51 (62.2%) were males and 31 (37.8%) were females. Mean age in study population was 60.26 ± 11.34 years. Majority of the population belongs to 56-65 years age group. The mean uric acid level of our study population was 6.03 ± 1.50 mg/dl (male = 5.92 ± 1.72, female = 6.64 ± 1.53). SUA ≥ 7 mg/dl was maximum in 56-65 years age group and there was no association between age and SUA (P value = 0.146). Over half of the study population were hypertensive i.e. 42 (51.21%) and smoker i.e. 43 (52.43%). It showed association between SUA and ACS (P value = 0.003). Among those having diabetes, maximum have SUA ≥ 7 mg/dl i.e. 17 (47.22%). Among those having high cholesterol level, male have higher incidence than female with no association between T. Cholesterol and gender (P value = 0.49). The mean value of T. Cholesterol was 189.83 ± 46.81 mg/dl (male = 198.78 ± 55.19 mg/dl, female = 202.30 ± 54.92 mg/dl) with (P value = 0.52). In conclusion, the mean age of ACS patients was 60.26 years, with the peak incidence at the age of 56-65 years. The ACS had male predominance. The potential risk factors of ACS were: Age >56 years (65.83%), male sex (62.2%), dyslipidemia (35.36%, hypertension (51.2%), diabetes mellitus (43.9%), smoking (52.4%) and alcohol consumption (39%). Among these SUA significantly associated with risk factors were- Sex, Diabetes Mellitus, T. Cholesterol. There was association between serum uric acid level and ACS patients. Hypertension and smoking constitutes one of the major risk factor for ACS in study population.  Full article
    References
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    Sharaf El Din UAA, Salem MM, Abdulazim DO. Uric acid in the pathogenesis of metabolic, renal, and cardiovascular diseases: A review. J Adv Res 2017;8(5): 537–548.
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    Maria Lorenza Muiesan, Claudia Agabiti-Rosei, Anna Paini, Massimo Salvetti. Uric Acid and Cardiovascular Disease: An Update. 2016;11(1):54–59.
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    Daskalopoulou SS, Athyros VG, Elisaf M, Mikhailidis DP. Uric acid levels and vascular disease. Curr Med Res Opin 2004;20(6):951-4.
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    Lehto S, Niskanen L, Rönnemaa T, Laakso M. Serum uric acid is a strong predictor of stroke in patients with non–insulin-dependent diabetes mellitus. Stroke 1998;29(3):635-9.
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    Kutzing MK, Firestein BL. Altered uric acid levels and disease states. J Pharmacol Exp Ther 2008;324(1):1-7.
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    Paudel B, Paudel K. Western Nepal acute coronary syndrome (WestNP-ACS) registry: Characteristics, management and in-hospital outcome of patients admitted with acute coronary syndrome in western Nepal. J GMC Nepal 2009;2(3):51-9.
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    Khatri P, Simkhada R. Study on conventional risk factors in acute coronary syndrome. J Universal College Med Sci. 2016;3(2):1-4.
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    Sinan Deveci O, Kabakci G, Okutucu S, Tulumen E, Aksoy H, Baris Kaya E, Evranos B, Aytemir K, Tokgozoglu L, Oto A. The association between serum uric acid level and coronary artery disease. Int J Clin Pract 2010;64(7):900-7.
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    Man KC, Rajbhandari S, Sharma D, Malla R, Rajbhandari R, Limbu Y, Regmi S, Maskey A. Distribution of Risk Factors in Patients with Acute Coronary Syndrome-A Hospital Based Study. J Nepal Med Assoc 2003;42(148);216-219.
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    Dali B. Clinical Profile, Dyslipidemia and ACS - a Correlation. J Nepal Med Assoc 2014;52(195):907-13.
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    Hasic S, Kadic D, Kiseljakovic E, Jadric R, Spahic E. Serum Uric Acid Could Differentiate Acute Myocardial Infarction and Unstable Angina Pectoris in Hyperuricemic Acute Coronary Syndrome Patients. Med Arch 2017;71(2):115-118.
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    Lim HE, Kim SH, Kim EJ, Kim JW, Rha SW, Seo HS, Park CG. Clinical value of serum uric acid in patients with suspected coronary artery disease. Korean J Intern Med 2010;25(1):21–26.
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